Longevity research · Est. 2025

Aging is a process.
Processes can be reversed.

Ananeo is a student-founded lab decoding why our cells grow old, and engineering ways to pause and reverse it.

01 — Assembly

Life begins as perfect order.

Billions of molecules fold, bind and settle into a living cell. Its information is intact. Its clock reads zero.

02 — Senescence

Then the signal starts to fade.

DNA damage piles up, epigenetic marks drift and mitochondria falter. The cell stops dividing but won't die. It goes grey.

03 — Intervention

We go in after the root cause.

Our molecules are built to find senescent cells, restore their energy and give back the instructions they lost.

04 — Renewal

The clock turns back.

Translucent, energetic, dividing again. We think aging can be treated like a disease, so we're working on a cure.

Scroll to age the cell

Why we exist

Every disease of old age, from cancer to Alzheimer's to heart failure, shares one risk factor: time. We think aging is a loss of information inside our cells, and lost information can be recovered. So we're not treating the symptoms one by one. We're going after the clock itself.

The 12 hallmarks of aging

Twelve ways a cell forgets how to be young.

Hover a card · 01/12

Interactive lab

Turn back the clock yourself.

Drag the slider to age the genome. Watch base pairs drift and break, colour fade and the helix come loose. Then hit rewind.

Biological age 20yrs
20406090
Telomere length11.0kb
NAD⁺ level100%
Senescent cells1.0%
Methylation drift0.00σ
Specimen · hTERT-23 State: youthful

Research pipeline

Three programs.
One clock.

A small-molecule combination that triggers apoptosis only in senescent cells, which pump out the inflammatory signals that age the tissue around them. Delivered as an oral capsule with a cyclical, hit-and-run dosing schedule.

  • Target · BCL-xL / p16INK4a cells
  • Model · aged murine fibroblasts
  • Readout · SASP markers (in vitro)

Transient, partial expression of reprogramming factors via lipid-nanoparticle mRNA, enough to restore a youthful epigenetic pattern without wiping the cell's identity.

  • Target · OSK factor cassette
  • Model · human dermal fibroblasts
  • Readout · Horvath clock age

An NAD⁺ precursor paired with a mitophagy activator. It clears out damaged mitochondria and fuels the new ones, so old cells get their power back.

  • Target · NAMPT / PINK1–Parkin axis
  • Model · C. elegans & aged mice
  • Readout · ATP output
0Hallmarks mapped
0Active programs
0Founders
0First-in-human target
Explore the full pipeline →

Be early

More time is the
only thing we all want.

Research updates, lab notes and early-access trials. No spam, ever.

Concept project by Tasos’s Studio · not a real companyConcept project · not a real company